In a biological quirk that promises to provide researchers with a new approach for studying and potentially treating Fragile X syndrome, scientists at the University of Massachusetts Medical School have shown that knocking out a gene important for messenger RNA translation in neurons restores memory deficits and reduces behavioral symptoms in a mouse model of a prevalent human neurological disease.
Fuente : http://www.eurekalert.org/pub_releases/2013-10/uom…
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